Publications with the contribution of LGSA PhD candidates
The goal of a PhD thesis program is to publish at least one first-author paper in an international peer-reviewed journal. We are very happy that our young researchers highly contribute to the publication record of the participating institutions.
2014
- Structure and biomedical applications of amyloid oligomer nanoparticles.
Kumar* ST, Meinhardt* J, Fuchs AK, Aumüller T, Leppert J, Büchele B, Knüpfer U, Ramachandran R, Yadav JK, Prell E, Morgado I, Ohlenschläger O, Horn U, Simmet T, Görlach** M, Fändrich** M
ACS NANO 2014, 8(11), 11042-52 * equal contribution, ** co-corresponding authors - Caspase-3 and Caspase-6 cleave STAT1 in leukemic cells.
Licht* V, Noack* K, Schlott B, Förster M, Schlenker Y, Licht A, Krämer OH, Heinzel T
Oncotarget 2014, 5(8), 2305-17 * equal contribution - Localisation microscopy with quantum dots using non-negative matrix factorisation.
Mandula O, Šestak IŠ, Heintzmann R, Williams CKI
Opt Express 2014, 22(20), 24594-605 - Long-term quiescent fibroblast cells transit into senescence.
Marthandan S, Priebe S, Hemmerich P, Klement K, Diekmann S
PLoS One 2014, 9(12), e115597 - Transporters MCT8 and OATP1C1 maintain murine brain thyroid hormone homeostasis.
Mayerl S, Müller J, Bauer R, Richert S, Kassmann CM, Darras VM, Buder K, Boelen A, Visser TJ, Heuer H
J Clin Invest 2014, 124(5), 1987–99 - Atherosclerosis-peripheral nervous system crosstalk in aged hyperlipidemic mice
Mohanta S
Dissertation 2014, Jena, Germany - Artery Tertiary Lymphoid Organs Contribute to Innate and Adaptive Immune Responses in Advanced Mouse Atherosclerosis.
Mohanta* SK, Yin* C, Peng L, Srikakulapu P, Bontha V, Hu D, Weih F, Weber C, Gerdes N, Habenicht AJR
Circ Res 2014, 114(11), 1772-87 * equal contribution - Tissue-Specific Alterations in Thyroid Hormone Homeostasis in Combined Mct10 and Mct8 Deficiency.
Müller J, Mayerl S, Visser TJ, Darras VM, Boelen A, Frappart L, Mariotta L, Verrey F, Heuer H
Endocrinology 2014, 155(1), 315-25 - The Tumor Suppressor PML Specifically Accumulates at RPA/Rad51-Containing DNA Damage Repair Foci but Is Nonessential for DNA Damage-Induced Fibroblast Senescence.
Münch S, Weidtkamp-Peters S, Klement K, Grigaravicius P, Monajembashi S, Salomoni P, Pandolfi PP, Weißhart K, Hemmerich P
Mol Cell Biol 2014, 34(10), 1733-46 - BOR-Syndrome-Associated Eya1 Mutations Lead to Enhanced Proteasomal Degradation of Eya1 Protein.
Musharraf A, Kruspe D, Tomasch J, Besenbeck B, Englert C, Landgraf K
PLoS One 2014, 9(1), e87407
