Laura Behrendt
Germany
LGSA Member
The role of membrane-shaping proteins in hereditary axonopathies
Friedrich Schiller University Jena (FSU): Faculty of Biological Sciences
Leibniz Institute on Aging - Fritz Lipmann Institute (FLI): Kaether Research Group
First Supervisor:
Christoph Kaether (Leibniz Institute on Aging - Fritz Lipmann Institute (FLI))Doctoral Disputation:
2021Publications 2023:
- A YIPF5-GOT1A/B complex directs a transcription-independent function of ATF6 in ER export
Cramer P, Yonemura Y, Behrendt L, Marszalek A, Sannai M, Durso W, Günes C, Szafranski K, Nakamura N, Nasrashvili T, Mayer J, von Eyss** B, Kaether** C
bioRxiv 2023, 10.1101/2023.12.12.569033 ** co-corresponding authors - Conserved exchange of paralog proteins during neuronal differentiation.
Di Fraia D, Anitei M, Mackmull MT, Parca L, Behrendt L, Andres-Pons A, Gilmour D, Helmer Citterich M, Kaether C, Beck M, Ori A
Life Sci Alliance 2022, 5(6) - The role of Atlastin-3 in hereditary axonopathies / by B.Sc., M.Sc., Laura Behrendt
Behrendt L
Dissertation 2021, Jena, Germany - Disease-causing mutated ATLASTIN 3 is excluded from distal axons and reduces axonal autophagy.
Behrendt L, Hoischen C, Kaether C
Neurobiol Dis 2021, 155, 105400 - Atlastine – wie defekte Netzwerke Neuropathien verursachen
Behrendt L, Kaether C
BIOspektrum 2020, 5, 485-7 - A disease causing ATLASTIN 3 mutation affects multiple endoplasmic reticulum-related pathways.
Behrendt L, Kurth I, Kaether C
Cell Mol Life Sci 2019, 76(7), 1433-45 - The nuclear pore proteins Nup88/214 and T-ALL-associated NUP214 fusion proteins regulate Notch signaling.
Kindermann B, Valkova C, Krämer A, Perner B, Engelmann C, Behrendt L, Kritsch D, Jungnickel B, Kehlenbach RH, Oswald F, Englert C, Kaether C
J Biol Chem 2019, 294(31), 11741-50